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Other secondary end points included the progression of renal disease and changes in the level of proteinuria.

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Similar reductions in mean ambulatory DBP and SBP were seen after treatment with olmesartan and irbesartan.

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The volume of distribution of losartan and the active metabolite is about 34 liters and 12 liters, respectively.

However, data are accumulating to support their use in patients with congestive heart failure, especially patients who cannot tolerate ACE inhibitors.

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Vasodilatation of arteriolar resistance vessels and venous capacitance vessels effectively unloads the heart, reduces LV wall stress, relieves coronary artery constriction, and redistributes flow through collaterals to reduce myocardial ischemia.

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Careful re-evaluation of the impact of stopping inhibitors of the renin-angiotensin system in patients with advanced chronic kidney disease.

In addition, we assessed the effects of losartan and placebo on the following secondary end points: a composite of morbidity and mortality from cardiovascular causes, proteinuria, and the rate of progression of renal disease.

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The bioavailability of the suspension formulation was compared with losartan tablets in healthy adults.

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Patients were randomized to receive losartan potassium 50 mg once daily or placebo on a background of conventional antihypertensive therapy excluding ACE inhibitors and angiotensin II antagonists.

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